Bluegrass Doctors of
Physical Therapy, PLLC
|Posted on December 30, 2015 at 9:55 AM|
My patients and I have had coutnless discussion of how even something as "tendonitis" can have central (IE central nervous system) effects in our body which can cause these ailments to perpetuate. This is often the case of my inteventions. I am trying to change processes that are in the CNS to then reduce pain in the periphery. Here is the abstract of an article that demonstrates just that with regrds to Achilles Tendonitis.
So I ask you, IS YOUR PRACTITIONER LOOKING AT CENTRAL MECHANISMS FOR YOUR PAIN?
Central pain processing is altered in people with Achilles tendinopathy
Background Tendinopathy is often a chronic condition. The mechanisms behind persistent tendon pain are not yet fully understood. It is unknown whether, similar to other persistent pain states, central pain mechanisms contribute to ongoing tendon pain.
Aim We investigated the presence of altered central pain processing in Achilles tendinopathy by assessing the conditioned pain modulation (CPM) effect in people with and without Achilles tendinopathy.
Methods 20 people with Achilles tendinopathy and 23 healthy volunteers participated in this cross-sectional study. CPM was assessed by the cold pressor test. The pressure pain threshold (PPT) was recorded over the Achilles tendon before and during immersion of the participant's hand into cold water. The CPM effect was quantified as the absolute difference in PPT before and during the cold pressor test.
Results An increase in PPT was observed in the Achilles tendinopathy and control group during the cold pressor test (p<0.001). However, the CPM effect was stronger in the control group (mean difference=160.5 kPa, SD=84.9 kPa) compared to the Achilles tendinopathy group (mean difference=36.4 kPa, SD=68.1 kPa; p<0.001).
Summary We report a reduced conditioned pain modulation effect in people with Achilles tendinopathy compared to people without Achilles tendinopathy. A reduced conditioned pain modulation effect reflects altered central pain processing which is believed to contribute to the persistence of pain in other conditions. Altered central pain processing may also be an important factor in persistent tendon pain that has traditionally been regarded to be dominated by peripheral mechanisms.